白癜风发病机制和治疗方法新进展(3)
[8]Birlea SA， Fain PR， Spritz RA. A Romanian population isolate with high frequency of vitiligo and associated autoimmune diseases. Archives of Dermatology， 2008， 144(3)：310.
[9]Natsch A. The Nrf2-Keap1-ARE toxicity pathway as a cellular sensor for skin sensitizers--functional relevance and a hypothesis on innate reactions to skin sensitizers. Toxicological Sciences An Official Journal of the Society of Toxicology， 2010， 113(2)：284.
[10]Denat L， Kadekaro AL， Marrot L， et al. Melanocytes as instigators and victims of oxidative stress. Journal of Investigative Dermatology， 2014， 134(6)：1512.
[11]Schallreuter KU， Salem MA， Gibbons NC， et al. Blunted epidermal L-tryptophan metabolism in vitiligo affects immune response and ROS scavenging by Fenton chemistry， part1： Epidermal H2O2/ONOO(-)-mediated stress abrogates tryptophan hydroxylase and dopa decarboxylase activities， leading to low serotonin and melatonin levels. FASEB J， 2012， 26(6)： 2457-2470.
[12]中國中西医结合学会皮肤性病专业委员会色素病学组. 白癜风诊疗共识(2014版). 中华皮肤科杂志， 2014， 47(1)：69-71.
[收稿日期：2017-12-20], http://www.100md.com(宋树玲)
[9]Natsch A. The Nrf2-Keap1-ARE toxicity pathway as a cellular sensor for skin sensitizers--functional relevance and a hypothesis on innate reactions to skin sensitizers. Toxicological Sciences An Official Journal of the Society of Toxicology， 2010， 113(2)：284.
[10]Denat L， Kadekaro AL， Marrot L， et al. Melanocytes as instigators and victims of oxidative stress. Journal of Investigative Dermatology， 2014， 134(6)：1512.
[11]Schallreuter KU， Salem MA， Gibbons NC， et al. Blunted epidermal L-tryptophan metabolism in vitiligo affects immune response and ROS scavenging by Fenton chemistry， part1： Epidermal H2O2/ONOO(-)-mediated stress abrogates tryptophan hydroxylase and dopa decarboxylase activities， leading to low serotonin and melatonin levels. FASEB J， 2012， 26(6)： 2457-2470.
[12]中國中西医结合学会皮肤性病专业委员会色素病学组. 白癜风诊疗共识(2014版). 中华皮肤科杂志， 2014， 47(1)：69-71.
[收稿日期：2017-12-20], http://www.100md.com(宋树玲)